Macrophages are exploited from an innate wound healing response to facilitate cancer metastasis.

Muliaditan, Tamara; Caron, Jonathan; Okesola, Mary; Opzoomer, James W; Kosti, Paris; Georgouli, Mirella; Gordon, Peter; Lall, Sharanpreet; Kuzeva, Desislava M; Pedro, Luisa; Shields, Jacqueline; Gillett, Cheryl E; Diebold, Sandra S; Sanz-Moreno, Victoria; Ng, Tony; Hoste, Esther; Arnold, James N

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Authors

Muliaditan, Tamara

Caron, Jonathan

Okesola, Mary

Opzoomer, James W

Kosti, Paris

Georgouli, Mirella

Gordon, Peter

Publication Date

2018-07-27

Journal Title

Nature communications

ISSN

2041-1723

Volume

Issue

Pages

2951

Language

eng

Type

Article

Physical Medium

Electronic

Metadata

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Citation

Muliaditan, T., Caron, J., Okesola, M., Opzoomer, J. W., Kosti, P., Georgouli, M., Gordon, P., et al. (2018). Macrophages are exploited from an innate wound healing response to facilitate cancer metastasis.. Nature communications, 9 (1), 2951. https://doi.org/10.1038/s41467-018-05346-7

Keywords

Cell Line, Tumor, Macrophages, Skin, Animals, Mice, Inbred BALB C, Humans, Mice, Mice, Mutant Strains, Breast Neoplasms, Neoplasm Metastasis, Collagen, Gelatinases, Serine Endopeptidases, Membrane Proteins, Interleukin-6, Cytokines, Prognosis, Wound Healing, Cell Survival, Gene Expression Regulation, Neoplastic, Phenotype, Female, Heme Oxygenase-1, Tumor Microenvironment

Sponsorship

MRC (MC_UU_12022/1_do not transfer?)

Medical Research Council (MC_UU_12022/5)

Identifiers

External DOI: https://doi.org/10.1038/s41467-018-05346-7

This record's URL: https://www.repository.cam.ac.uk/handle/1810/284694

Rights

Attribution 4.0 International

Licence URL: https://creativecommons.org/licenses/by/4.0/

Except where otherwise noted, this item's licence is described as Attribution 4.0 International