Norovirus infection results in eIF2α independent host translation shut-off and remodels the G3BP1 interactome evading stress granule formation.

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Authors
Brocard, Michèle
Iadevaia, Valentina
Klein, Philipp
  Hall, Belinda
Lewis, Glenys
  Lu, Jia
Burke, James
Publication Date
2020-01-06
Journal Title
PLoS pathogens
ISSN
1553-7366
Volume
16
Issue
1
Pages
e1008250
Language
eng
Type
Article
This Version
VoR
Physical Medium
Electronic-eCollection
Metadata
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Citation
Brocard, M., Iadevaia, V., Klein, P., Hall, B., Lewis, G., Lu, J., Burke, J., et al. (2020). Norovirus infection results in eIF2α independent host translation shut-off and remodels the G3BP1 interactome evading stress granule formation.. PLoS pathogens, 16 (1), e1008250. https://doi.org/10.1371/journal.ppat.1008250
Keywords
Cell Line, Cytoplasmic Granules, Animals, Mice, Inbred C57BL, Humans, Mice, Norovirus, Caliciviridae Infections, DNA Helicases, RNA Helicases, Eukaryotic Initiation Factor-2, RNA, Virus Replication, Signal Transduction, Protein Biosynthesis, RAW 264.7 Cells, RNA Recognition Motif Proteins, Poly-ADP-Ribose Binding Proteins
Sponsorship
BBSRC (BB/N001176/1)
Wellcome Trust (207498/Z/17/Z)
Identifiers
External DOI: https://doi.org/10.1371/journal.ppat.1008250
This record's URL: https://www.repository.cam.ac.uk/handle/1810/301556

Rights
Attribution 4.0 International
Licence URL: https://creativecommons.org/licenses/by/4.0/

Attribution 4.0 International
Except where otherwise noted, this item's licence is described as Attribution 4.0 International