Norovirus infection results in eIF2α independent host translation shut-off and remodels the G3BP1 interactome evading stress granule formation.
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Authors
Brocard, Michèle
Iadevaia, Valentina
Klein, Philipp
Lewis, Glenys
Burke, James
Willcocks, Margaret M
Parker, Roy
Publication Date
2020-01-06Journal Title
PLoS pathogens
ISSN
1553-7366
Volume
16
Issue
1
Pages
e1008250
Language
eng
Type
Article
This Version
VoR
Physical Medium
Electronic-eCollection
Metadata
Show full item recordCitation
Brocard, M., Iadevaia, V., Klein, P., Hall, B., Lewis, G., Lu, J., Burke, J., et al. (2020). Norovirus infection results in eIF2α independent host translation shut-off and remodels the G3BP1 interactome evading stress granule formation.. PLoS pathogens, 16 (1), e1008250. https://doi.org/10.1371/journal.ppat.1008250
Keywords
Cell Line, Cytoplasmic Granules, Animals, Mice, Inbred C57BL, Humans, Mice, Norovirus, Caliciviridae Infections, DNA Helicases, RNA Helicases, Eukaryotic Initiation Factor-2, RNA, Virus Replication, Signal Transduction, Protein Biosynthesis, RAW 264.7 Cells, RNA Recognition Motif Proteins, Poly-ADP-Ribose Binding Proteins
Sponsorship
BBSRC (BB/N001176/1)
Wellcome Trust (207498/Z/17/Z)
Identifiers
External DOI: https://doi.org/10.1371/journal.ppat.1008250
This record's URL: https://www.repository.cam.ac.uk/handle/1810/301556