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dc.contributor.authorFrasch, Martin G
dc.contributor.authorGiussani, Dino A
dc.date.accessioned2020-04-21T00:24:27Z
dc.date.available2020-04-21T00:24:27Z
dc.date.issued2020-03-17
dc.identifier.issn2073-4409
dc.identifier.otherPMC7140710
dc.identifier.other32192015
dc.identifier.urihttps://www.repository.cam.ac.uk/handle/1810/304566
dc.description.abstractChronic fetal hypoxia and infection are examples of adverse conditions during complicated pregnancy, which impact cardiac myogenesis and increase the lifetime risk of heart disease. However, the effects that chronic hypoxic or inflammatory environments exert on cardiac pacemaker cells are poorly understood. Here, we review the current evidence and novel avenues of bench-to-bed research in this field of perinatal cardiogenesis as well as its translational significance for early detection of future risk for cardiovascular disease.
dc.languageeng
dc.rightsAttribution 4.0 International
dc.rights.urihttps://creativecommons.org/licenses/by/4.0/
dc.sourceessn: 2073-4409
dc.sourcenlmid: 101600052
dc.subjectCardiac development
dc.subjectSinus Node
dc.subjectFetal Programming
dc.subjectIhrv
dc.subjectIntrinsic Heart Rate Variability
dc.titleImpact of Chronic Fetal Hypoxia and Inflammation on Cardiac Pacemaker Cell Development.
dc.typeArticle
dc.date.updated2020-04-21T00:24:26Z
prism.issueIdentifier3
prism.publicationNameCells
prism.volume9
dc.identifier.doi10.17863/CAM.51648
rioxxterms.versionofrecord10.3390/cells9030733
rioxxterms.versionVoR
rioxxterms.licenseref.urihttps://creativecommons.org/licenses/by/4.0/
dc.contributor.orcidFrasch, Martin G [0000-0003-3159-6321]
dc.contributor.orcidGiussani, Dino A [0000-0002-1308-1204]


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Attribution 4.0 International
Except where otherwise noted, this item's licence is described as Attribution 4.0 International