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dc.contributor.authorGill, Dipender
dc.contributor.authorBurgess, Stephen
dc.date.accessioned2022-03-07T02:04:31Z
dc.date.available2022-03-07T02:04:31Z
dc.date.issued2022-02
dc.identifier.issn1549-1277
dc.identifier.otherPMC8812877
dc.identifier.other35113864
dc.identifier.urihttps://www.repository.cam.ac.uk/handle/1810/334718
dc.description.abstractDipender Gill and Stephen Burgess discuss the accompanying study by James Yarmolinsky and colleagues investigating the associations between genetically-proxied inhibition of antihypertensive drug targets and risk of common cancer subtypes using Mendelian randomization.
dc.languageeng
dc.publisherPublic Library of Science (PLoS)
dc.rightsAttribution 4.0 International
dc.rights.urihttps://creativecommons.org/licenses/by/4.0/
dc.sourcenlmid: 101231360
dc.sourceessn: 1549-1676
dc.subjectAngiotensin-Converting Enzyme Inhibitors
dc.subjectAntihypertensive Agents
dc.subjectBlood Pressure
dc.subjectColorectal Neoplasms
dc.subjectGenetic Variation
dc.subjectGenome-Wide Association Study
dc.subjectHumans
dc.subjectMendelian Randomization Analysis
dc.subjectRisk Factors
dc.titleThe evolution of mendelian randomization for investigating drug effects.
dc.typeArticle
dc.date.updated2022-03-07T02:04:31Z
prism.issueIdentifier2
prism.publicationNamePLoS Med
prism.volume19
dc.identifier.doi10.17863/CAM.82136
dcterms.dateAccepted2022-02-03
rioxxterms.versionofrecord10.1371/journal.pmed.1003898
rioxxterms.versionVoR
rioxxterms.licenseref.urihttps://creativecommons.org/licenses/by/4.0/
dc.contributor.orcidGill, Dipender [0000-0001-7312-7078]
dc.contributor.orcidBurgess, Stephen [0000-0001-5365-8760]
dc.identifier.eissn1549-1676
pubs.funder-project-idWellcome Trust (204623/Z/16/Z)
pubs.funder-project-idMedical Research Council (MC_UU_00002/7)
cam.issuedOnline2022-02-03


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Attribution 4.0 International
Except where otherwise noted, this item's licence is described as Attribution 4.0 International