Research data supporting "Maternal diet-induced obesity during pregnancy alters lipid supply to mouse E18.5 fetuses and changes the cardiac tissue lipidome in a sex-dependent manner"
Citation
Carminatti Pantaleao, L., & Inzani, I. (2022). Research data supporting "Maternal diet-induced obesity during pregnancy alters lipid supply to mouse E18.5 fetuses and changes the cardiac tissue lipidome in a sex-dependent manner" [Dataset]. https://doi.org/10.17863/CAM.80205
Description
The xlsx file contains a collection of datasets characterizing the cardiac phenotype and the cardiac and circulatory lipid profiles in late gestation E18.5 fetuses of diet-induced obese pregnant mice.
These several datasets contain the processed data used in the article "Maternal diet-induced obesity during pregnancy alters lipid supply to mouse E18.5 fetuses and changes the cardiac tissue lipidome in a sex-dependent manner", published on eLife (doi: 10.7554/eLife.69078)
Datasets are separated in tabs with a brief description of the data, as well as outlier allocation and criteria.
Format
R v.4.1.2
Ingenuity Pathways Analysis (IPA) v.42012434
edgeR v.3.36.0
QuPath v.0.3.0
Xcalibur v.4.3
Harmony v5.0
ImageJ v.1.53n
XCMS v.3.16.1
Peakpicker v.2.0
Keywords
Maternal obesity, Fetal heart, Lipidomics, Heart metabolism
Relationships
Publication Reference: https://doi.org/10.7554/eLife.69078https://www.repository.cam.ac.uk/handle/1810/333744
Sponsorship
This work was funded by the British Heart Foundation [RG/17/12/33167], the MRC Metabolic Diseases Unit [MRC_MC_UU_00014/4], and the Wellcome Trust [208363/Z/17/Z]. A.A.M.C was supported by a Cambridge Home and EU Student Scholarship. E.L. was supported by a British Heart Foundation studentship [FS/12/64/30001]. II was supported by a British Heart Foundation studentship [FS/18/56/35177]. S.F. was supported by the BBSRC [BB/M027252/1].
Identifiers
This record's DOI: https://doi.org/10.17863/CAM.80205
Rights
Attribution 4.0 International (CC BY 4.0)
Licence URL: https://creativecommons.org/licenses/by/4.0/
Statistics
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