Nutrition for Gestational Diabetes-Progress and Potential


Type
Article
Change log
Authors
Abstract

Gestational diabetes (GDM), traditionally defined as any form of glucose intolerance first detected in pregnancy [1], still has dietary treatment as its frontline therapy [2]. Whilst some women require additional pharmacotherapy, such as with insulin or metformin, nutritional intake is relevant to all women with GDM. Recognition of the importance of GDM is growing due to the worldwide increase in its prevalence [3], rising in line with the increased prevlance of overweight and obesity [4], and the impact of its short- and long-term complications in both the mother and offspring exposed to GDM in utero [5]. In fact it has been suggested that GDM makes a significant contribution to the current diabetes epidemic [6]. This partially relates to the increased risk of obesity [7], insulin resistance [8], GDM [9] and type 2 diabetes [10] in people who were exposed to GDM in fetal life, showing that “diabetes begets diabetes” [11]. Thus preventing and treating GDM more efficiently have a sense of urgency about them, with nutritional modifications needing to be at the forefront. This is especially since a recent keynote systematic review and meta-analysis of nutritional interventions in GDM that were tested in randomized, controlled trials found favourable effects on maternal glycemic control and neonatal growth parameters when grouped together [12]. It is with these considerations that this Special Issue of Nutrients has been published, looking at nutrients or factors related to nutrition that may be involved either in the prevention, development or treatment of GDM. In this way I hope that it plays some part in showing current progress and the potential for future improvements in nutrition for GDM.

Description
Keywords
Diabetes, Gestational, Female, Gastrointestinal Microbiome, Glucose Intolerance, Humans, Insulin Resistance, Nutritional Status, Obesity, Pregnancy
Journal Title
Nutrients
Conference Name
Journal ISSN
2072-6643
2072-6643
Volume Title
Publisher
MDPI AG
Sponsorship
None