Research data supporting "SGK1 mutations in DLBCL generate hyperstable protein neoisoforms that promote AKT independence" (Supplementary tables 1 and 2)
Table S1. List of all SGK1 variants identified across five DLBCL sequencing studies used to generate Figure 1A & B. Table S2. Summary of Intron-1 retention scoring from analysis of RNA-seq data from the Goya trial in patients with and without the indicated splice mutations.