Research data supporting "SGK1 mutations in DLBCL generate hyperstable protein neoisoforms that promote AKT independence" (Supplementary tables 1 and 2)

Name: Research data supporting "SGK1 mutations in DLBCL generate hyperstable protein neoisoforms that promote AKT independence" (Supplementary tables 1 and 2)
Published: 2021-05-14T14:21:37Z
Keywords: Stem Cell Institute

Hodson, Daniel

doi:10.17863/CAM.69799

Research data supporting "SGK1 mutations in DLBCL generate hyperstable protein neoisoforms that promote AKT independence" (Supplementary tables 1 and 2)

Repository URI

https://www.repository.cam.ac.uk/handle/1810/322397

Repository DOI

https://doi.org/10.17863/CAM.69799

Files

Supplementary Table 1.xlsx (59.03 KB)

Supplementary Table 2.xlsx (23.77 KB)

Type

Dataset

Authors

Hodson, Daniel

https://orcid.org/0000-0001-6225-2033

Description

Table S1. List of all SGK1 variants identified across five DLBCL sequencing studies used to generate Figure 1A & B. Table S2. Summary of Intron-1 retention scoring from analysis of RNA-seq data from the Goya trial in patients with and without the indicated splice mutations.