Targeting adhesion to the vascular niche to improve therapy for acute myeloid leukemia
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Peer-reviewed
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Abstract
Niche hijack by malignant cells is considered to be a prominent cause of disease relapse. Barbier and colleagues uncover (E)-selectin as a novel mediator of malignant cell survival and regeneration which, upon blockade, has the potential to significantly improve therapeutic outcomes.
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Comment, /692/4028/67/327, /692/4028/67/1059/2326, /13/31, /64/60, /13/106, comment
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Nature Publishing Group UK
