Targeting adhesion to the vascular niche to improve therapy for acute myeloid leukemia
Published version
Peer-reviewed
Repository URI
Repository DOI
Change log
Authors
Abstract
Niche hijack by malignant cells is considered to be a prominent cause of disease relapse. Barbier and colleagues uncover (E)-selectin as a novel mediator of malignant cell survival and regeneration which, upon blockade, has the potential to significantly improve therapeutic outcomes.
Description
Keywords
Is Part Of
Publisher
Nature Publishing Group UK
Publisher DOI
Rights and licensing
Except where otherwised noted, this item's license is described as http://creativecommons.org/licenses/by/4.0/