This repository holds the research output of members of the University of Cambridge. It is delivered and managed by the University Library's Office of Scholarly Communication team.

  • Single cell analysis reveals human cytomegalovirus drives latently infected cells towards an anergic-like monocyte state 

    Shnayder, Miri; Nachshon, Aharon; Rozman, Batsheva; Bernshtein, Biana; Lavi, Michael; Fein, Noam; Poole, Emma et al. (eLife Sciences Publications, Ltd, 2020-01-22)
    Human cytomegalovirus (HCMV) causes a lifelong infection through establishment of latency. Although reactivation from latency can cause life-threatening disease, our molecular understanding of HCMV latency is incomplete. ...
  • The global variation of medical student engagement in teaching: Implications for medical electives 

    Wenlock, Rhys D.; Bath, Michael F.; Bashford, Tom; Kohler, Katharina; Hutchinson, Peter J. (Public Library of Science, 2020-02-24)
    Introduction: International medical electives, whereby undergraduates visit an institution in a country other than their own, are a common part of medical training. Visiting students are often asked to provide local teaching, ...
  • Forecasting the prevalence of overweight and obesity in India to 2040 

    Luhar, Shammi; Timæus, Ian M.; Jones, Rebecca; Cunningham, Solveig; Patel, Shivani A.; Kinra, Sanjay; Clarke, Lynda et al. (Public Library of Science, 2020-02-24)
    Background: In India, the prevalence of overweight and obesity has increased rapidly in recent decades. Given the association between overweight and obesity with many non-communicable diseases, forecasts of the future ...
  • Loss of Kat2a enhances transcriptional noise and depletes acute myeloid leukemia stem-like cells 

    Domingues, Ana Filipa; Kulkarni, Rashmi; Giotopoulos, George; Gupta, Shikha; Vinnenberg, Laura; Arede, Liliana; Foerner, Elena et al. (eLife Sciences Publications, Ltd, 2020-01-27)
    Acute Myeloid Leukemia (AML) is an aggressive hematological malignancy with abnormal progenitor self-renewal and defective white blood cell differentiation. Its pathogenesis comprises subversion of transcriptional regulation, ...

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