Research data supporting 'Activation of the proton-sensing GPCR, GPR65 on fibroblast-like synoviocytes contributes to inflammatory joint pain.'

Name: Research data supporting 'Activation of the proton-sensing GPCR, GPR65 on fibroblast-like synoviocytes contributes to inflammatory joint pain.'
Published: 2024-11-22T09:22:41Z
Keywords: gpcr, nociception, pharmacology

Pattison, Luke

doi:https://doi.org/10.17863/CAM.106374

Research data supporting 'Activation of the proton-sensing GPCR, GPR65 on fibroblast-like synoviocytes contributes to inflammatory joint pain.'

Repository URI

https://www.repository.cam.ac.uk/handle/1810/364830

Repository DOI

https://doi.org/10.17863/CAM.106374

Files

Fig1_data.csv (24.92 KB)

Fig2_data.csv (13.8 KB)

Fig3TableS1TableS2_data.csv (9.7 KB)

Fig4TableS3TableS4_data.csv (21.94 KB)

Fig5TableS5TableS6_data.csv (16.36 KB)

Type

Dataset

Authors

Pattison, Luke

https://orcid.org/0000-0001-8571-3314

Description

Data underlying the figures from the associated paper "Activation of the proton-sensing GPCR, GPR65 on fibroblast-like synoviocytes contributes to inflammatory joint pain".

Each .csv file contains the data constituting the Figures and Tables referenced in the file name.

An explanation of how data under each column was collected/analysed can be found in the header_info.csv.

Software / Usage instructions

Curated datasets exported from R.

Keywords

gpcr, nociception, pharmacology

Rights and licensing

Except where otherwised noted, this item's license is described as Attribution-NonCommercial-NoDerivatives 4.0 International (CC BY-NC-ND 4.0)

Sponsorship

BBSRC (1943916)
Biotechnology and Biological Sciences Research Council (1943916)
BBSRC (1943916)
MRC (MR/W002426/1)
BBSRC (BB/V509528/1)
Wellcome Trust (225856/Z/22/Z)

BBSRC Doctoral Training Programme (BB/MO11194/1) Corpus Christi College MRC (MR/W002426/1 & MR/W026961/1) AstraZeneca (G104108) BBSRC / GSK (BB/V509528/1) Wellcome Trust (225856/Z/22/Z) Versus Arthritis (21530) Royal Society (NF\R2\212001) From the paper: The authors acknowledge the following sources of funding to support this work: University of Cambridge BBSRC Doctoral Training Programme BB/MO11194/1 (LAP); Corpus Christi College, University of Cambridge Studentship (LAP); MRC Grant MR/W002426/1 (LAP, ESS); AstraZeneca PhD Studentship G104108 (RHR); BBSRC/GSK iCASE PhD Studentship BB/V509528/1 (HH); Wellcome Trust Grant 225856/Z/22/Z (MD, ESS); MRC Grant MR/W026961/1 (SNW, SWJ); Versus Arthritis Grant 21530 (SNW, SWJ); Royal Society Industry Fellowship NF\R2\212001 (GL).