Aberrant anaplastic lymphoma kinase activity induces a p53 and Rb-dependent senescence-like arrest in the absence of detectable p53 stabilization.

Skerrett, Fiona McDuff; Turner, Suzanne

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Authors

Skerrett, Fiona McDuff

Turner, Suzanne

Publication Date

2011-03-14

Journal Title

PloS one

ISSN

1932-6203

Volume

Issue

Pages

e17854

Language

eng

Type

Article

This Version

VoR

Physical Medium

Electronic

Metadata

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Citation

Skerrett, F. M., & Turner, S. (2011). Aberrant anaplastic lymphoma kinase activity induces a p53 and Rb-dependent senescence-like arrest in the absence of detectable p53 stabilization.. PloS one, 6 (3), e17854. https://doi.org/10.1371/journal.pone.0017854

Keywords

Cells, Cultured, Fibroblasts, Animals, Humans, Mice, Cell Transformation, Neoplastic, Receptor Protein-Tyrosine Kinases, Retinoblastoma Protein, Cell Adhesion, Cell Cycle, Cell Proliferation, Transcription, Genetic, Protein Processing, Post-Translational, Mutation, Models, Biological, Tumor Suppressor Protein p53, Cyclin-Dependent Kinase Inhibitor p16, Protein-Tyrosine Kinases, Embryo, Mammalian, Protein Stability, Cellular Senescence, Anaplastic Lymphoma Kinase

Identifiers

External DOI: https://doi.org/10.1371/journal.pone.0017854

This record's URL: https://www.repository.cam.ac.uk/handle/1810/305800

Rights

Attribution 4.0 International

Licence URL: https://creativecommons.org/licenses/by/4.0/

Except where otherwise noted, this item's licence is described as Attribution 4.0 International